Designed Around How Parkinson's Affects Movement.
Parkinson's disease progressively impairs the brain's ability to initiate and sustain voluntary muscle contraction. Dopamine depletion within the basal ganglia disrupts motor sequencing, reducing the reliability of voluntary muscle activation and weakening communication between neural intent and physical movement. The brain transmits a signal. The body stops receiving it clearly.
The Stabilis NMES Therapeutic Suit is designed in direct response to this disruption — using therapeutic muscle stimulation to support mobility, stability, and muscle activation while reinforcing communication between the brain, spine, and body. Through targeted activation of muscle fibers and consistent therapeutic engagement, the platform is designed to support movement function and promote healthier neuromuscular activity over time.
Direct activation.
Without the detour.
The suit bypasses the compromised pathway. Directly.
The suit supports movement by helping reinforce compromised neuromuscular pathways through targeted therapeutic stimulation.
In healthy motor function, the brain sends signals through the corticospinal tract to recruit muscle activity. In Parkinson's disease, dopamine depletion within the basal ganglia disrupts motor sequencing, reducing the reliability of voluntary muscle activation and weakening communication between neural intent and physical movement.
The Stabilis NMES Therapeutic Suit is designed in direct response to this disruption. Targeted electrical stimulation activates motor nerve branches and muscle fibers to support muscle contraction, mobility, and stability — reinforcing communication between the brain, spine, and body through consistent therapeutic engagement.
Physician prescribed for home use and monitored remotely through connected sensor technology, the platform continuously adapts stimulation intensity based on movement patterns and therapeutic progress over time.
The mechanism
Three mechanisms. One session.
Each session targets the neuromuscular disruptions associated with Parkinson's disease across three interconnected therapeutic mechanisms.
Involuntary contraction without external load
NMES generates full muscle recruitment via direct electrical activation — no voluntary initiation required, no added weight or resistance. Patients achieve the neuromuscular output that Parkinson's disease progressively prevents. The muscle contracts fully, even when the brain's motor command chain cannot reliably produce that instruction.
Rebuilding the neuromuscular connection
Each stimulation-assisted contraction reinforces the neural pathway between motor neuron and muscle fiber. Repeated activation over weeks drives activity-dependent neuroplasticity — progressively improving the nervous system's ability to voluntarily recruit muscle alongside the electrically-generated response.
Stimulation that adapts to you
Onboard sensor fusion monitors movement quality in real time. When movement quality decreases, stimulation intensity increases to compensate. As functional capacity builds, the system scales back. Every session is calibrated to where the patient is today — not where they were last week.
Patient experiences.
Personal experiences from patients using the Stabilis NMES Therapeutic Suit throughout the therapeutic program.
"Participating in the workouts helped me become more aware of my physical limitations. The suit was comfortable to wear, and even after it was turned off, I felt that the stimulation effects continued — making it easier for me to move."
"As I progressed through the program, my morning routine changed. I found I could delay my first dose by an hour and a half each morning — and I was going 8 to 9 hours without medication from morning to evening."
4.8 out of 5 patient satisfaction score · 40+ participants · 12-week pilot trial
What the suit knows about you.
A central near-infrared light panel delivers targeted PBM during and after each session — stimulating mitochondrial activity to accelerate muscle recovery and reduce post-session fatigue.
Distributed IMUs track orientation, velocity, and movement patterns in real time. The suit classifies exercise execution automatically — confirming correct form and adjusting stimulation intensity to match.
Bioimpedance sensors assess body composition at regular intervals throughout the program — tracking muscle hydration and quality over time to inform how stimulation adapts across sessions.
Near-infrared spectroscopy monitors heart rate and tissue oxygen saturation continuously. The suit tracks physiological load in real time, ensuring stimulation stays within safe and effective parameters.
Impedance sensors confirm that every electrode is properly seated before stimulation begins. If contact is lost mid-session, stimulation stops immediately — automatically.
The suit separates at the waist into an upper vest and lower component. Each piece is put on independently while loose, then zipped to a secure, fitted position once on the body.
Zippers at every extremity and the waist allow full entry while loose. Singer loop pull tabs throughout give a secure grip point for pulling the garment firmly over the arms and legs.
Before every session, the suit runs an automated impedance check across all electrodes. Stimulation only begins when full contact is confirmed — no manual setup, no guesswork.
Stimulation intensity ramps up progressively at session start — no sudden activation. If electrode contact is lost at any point, the suit stops immediately and automatically.
Over time
What changes with consistent use.
The nervous system adapts.
Activity dependent neuroplasticity allows therapeutic gains to begin carrying over beyond individual therapy sessions. As the nervous system is repeatedly challenged through stimulation assisted contractions, voluntary motor function begins to strengthen alongside therapeutic activation. Over time, improvements in movement quality, stability, and motor control can begin to translate more naturally into everyday life.
Getting dressed without overthinking every movement.
Holding a glass with confidence and control.
Moving through your home more naturally again.
Keeping up with the people you love.
Ready to learn more?
Is this therapy right for you?
Designed for Parkinson's patients and prescribed for home use, with remote monitoring and therapy tailored to your progress over time.
Peer-Reviewed References
- Maffiuletti, N.A. (2010). Physiological and methodological considerations for the use of neuromuscular electrical stimulation. European Journal of Applied Physiology, 110(2), 223–234.
- Veldman, M.P. et al. (2016). Direct and crossed effects of somatosensory stimulation on neuronal excitability and motor performance. Neuroscience & Biobehavioral Reviews, 68, 260–276.
- Paul, S.S. et al. (2014). Characteristics of falls in people with Parkinson's disease: implications for fall prevention. Journal of Neurology, Neurosurgery & Psychiatry, 85(6), 596–600.
- Allen, N.E. et al. (2011). The effects of an exercise program on fall risk factors in people with Parkinson's disease: a randomized controlled trial. Movement Disorders, 26(8), 1484–1491.
